Efficacy of antibiotics questioned as Colistin also termed ineffective
In the beginning of April, experts at a military lab outside Washington strengthened their hunt for proof that an unsafe new biological threat had crossed the nation’s borders. They didn’t require looking for it much as they found it in no time.
On May 18, a team from the Walter Reed Army Institute of Research got its first glance at a sample of the bacterium Escherichia coli, collected form a 49-year-old Pennsylvania woman. She suffered from a urinary tract infection having a disturbing ability for surviving the assaults of antibiotic medications. Her sample was among the six countrywide being given to the lab of microbiologist Patrick McGann.
Soon, a preliminary study deepened worries at the lab. For the following some days, more sophisticated genetic sleuthing verified the worst fears of McGann.
They found a gene, dubbed mcr-1, in the bacterium’s DNA. Its presence made the pathogen resistant to the esteemed antibiotic colistin.
More worrisome was the fact that the gene was found on a plasmid, a small mobile loop of DNA that can be readily snapped off and attached to other bacteria, suggesting that it may readily jump to other E. coli bacteria, or to completely distinct kinds of disease-causing organisms. In case it happens, it would make them unreceptive to colistin as well.
The public health officials were looking forward to this milestone for years. In a firm march, disease-causing microbes have found ways to avoid the protection of medications used in the treatment of bacterial infections.
For a variety of such illnesses, just colistin proved effective every time, and now the last line of defense had also got breached.
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